Skip Navigation

This Article
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (13)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Stirling, R G
Right arrow Articles by Chung, K F
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Stirling, R G
Right arrow Articles by Chung, K F
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

British Medical Bulletin 56:1037-1053 (2000)
© 2000 The British Council


research-article

Future treatments of allergic diseases and asthma

R G Stirling and K F Chung

National Heart & Lung Institute, Imperial College School of Medicine and Royal Brompton Hospital London, UK

Correspondence to:Professor Fan Chung, Departments of Thoracic Medicine, National Heart & Lung Institute, Dovehouse Street, London SW3 6LY, UK

Abstract

Recent advances in the understanding of the inflammatory and immunological mechanisms of allergic diseases have illuminated many potential therapeutic stratigies that may prevent or even reverse the abnormalities of allergic inflammation. As the roles of effector cells, and of signalling and adhension molecules are better understood, the opportunities to inhibit or prevent the inflammatory cascade have increased. In addition, there have been advances in the synthesis of proteins, monoclonal antibodies and new small molecule chemical entities, which provide further valuable flexibility in the therapeutic appraoch to asthma. Such new approaches are aimed at prevention of T-cell activation; redressing the imbalance of T helper cell populations thus inhibiting or preventing Th-2-derived cytokine expression; and the inhibition or blockade of the downstream actions of these cytokiness such as effects on LqE and eosinophils. Approaches such as these allow both broad and highly specific targeting, and may pave the way towards the prevention and reversal of the immunological and inflammatory processes driving asthma, allergic rhinitis and atopic dermatitis. The development of effective agents with effects beyound those provided by current therapies coupled with lesser side-effects will further address the unmet needs of allergic disease.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?




Disclaimer:
Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.